Atlas of Clinical Hematology - part 5 pdf
Atlas of Clinical Hematology potx
... Dr. Witte of Erlangen,Dr. Langreder of Mainz, Prof. Dr. Mohr of the Tropical Institute of Hamburg,Dr. Moeschlin of Zurich, Dr. Undritz of Basel, and Dr. Kuhn of our FreiburgClinic. We also thank ... We also thankthe Institute of Pathology of the University of Kiel, headed by Prof. Karl Len-nert, and the current head of the Department of Hematologic Pathology,Prof. Reza Parwaresch, for preparing ... sections of the tissue coresthat we submitted.AcknowledgementsWe are indebted to Prof. Brigitte Schlegelberger, Prof. Werner Grote (direc-tor of the Institute of Human Genetics, University of Kiel),...
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Chapter 005. Principles of Clinical Pharmacology (Part 5) doc
... effect of the drugs, it may produce severe agonist–mediated effects (such as hypertension or tachycardia) if the blocking drug is abruptly withdrawn. Chapter 005. Principles of Clinical Pharmacology ... site of action, it alters the function of that molecular target, with the ultimate result of a drug effect that the patient or physician can perceive. For drugs used in the urgent treatment of ... accumulation of active metabolites. Commonly, the clinical effect develops as a downstream consequence of the initial molecular effect the drug produces. Thus, administration of a proton-pump...
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Chapter 005. Principles of Clinical Pharmacology (Part 6) pdf
... Response Chapter 005. Principles of Clinical Pharmacology (Part 6) Principles of Dose Selection The desired goal of therapy with any drug is to maximize the likelihood of a beneficial ... an understanding of the relationships between plasma concentration and anticipated effects. For example, measurement of QT interval is used during treatment with sotalol or dofetilide to avoid ... concentration and beneficial or adverse drug effect, measurement of plasma levels can provide a valuable tool to guide selection of an optimal dose. This is particularly true when there is a...
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Chapter 105. Malignancies of Lymphoid Cells (Part 5) pdf
... Chapter 105. Malignancies of Lymphoid Cells (Part 5) Table 105-5 Diseases or Exposures Associated with Increased Risk of Development of Malignant Lymphoma Inherited ... activation of a series of transcription factors, the cell first becomes committed to the lymphoid lineage and then gives rise to B and T cells. About 75% of all lymphoid leukemias and 90% of all ... chemotherapy and radiation therapy Immunology All lymphoid cells are derived from a common hematopoietic progenitor that gives rise to lymphoid, myeloid, erythroid, monocyte, and megakaryocyte...
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Chapter 127. Treatment and Prophylaxis of Bacterial Infections (Part 5) pdf
... permeability and cause active drug efflux from the cytoplasm. Mutations that result in active quinolone efflux are also found in gram-positive bacteria. Chapter 127. Treatment and Prophylaxis of Bacterial ... dihydrofolate reductase for trimethoprim and an altered dihydropteroate synthetase for sulfonamides. Quinolones The most common mechanism of resistance to quinolones is the development of one ... The most prevalent mechanism of resistance to trimethoprim and the sulfonamides in both gram-positive and gram-negative bacteria is the acquisition of plasmid-encoded genes that produce...
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Atlas of Clinical Hematology - part 1 ppsx
... Reaction) 11 1. 11 Sudan Black B Stain 13 1. 12 Cytochemical Determination of Peroxidase 13 1. 13 Hydrolases 13 1. 13 .1 Cytochemical Determination of Leukocyte Alkaline Phosphatase (LAP)in Blood Smears 13 1. 13.2 ... CausativeOrganisms of Tropical Disease 399 IITable 2. Cytochemistry of blood and bone marrow cellsPer-oxidasePAS Esterasea-Naphthyl-acetate-,Naphthol-AS-acetate-Naphthol-AS-D-chloracetate-Phosphatasesalkaline ... formalin (10 : 1) . DAB solution: 5 mg diaminobenzidine tetrahy-drochloride in 20 mL of 0.05 mol/L tris-HClbuffer (pH 7.6) with 50 lLof1%H2O2added Tris-HCl: 50 mL of solution A (12 1 .14 g...
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Atlas of Clinical Hematology - part 2 pot
... pro-ceeding with a more detailed evaluation.Figure 22 f, g shows the appearance of a normalbone marrow smear viewed at high magnifica-tion. Figure 22 a, b illustrates the range of varia-tion ... (histologic sec-tions). Figure 22 c shows how the naphthol AS-Dchloroacetate esterase method can be used to lo-calize early neutrophilic granulocytop oiesis closeto bony trabeculae. Figure 22 d, e ... myeloidleukemiaChronic lympho-cytic leukemiaAgranulocytosis 20 40 60 80 20 40 60 80 20 40 60 80 20 40 60 80 20 40 60 80 20 40 60 80 20 40 60 80 20 40 6080Reticulum cellsPlasma cellsMegaloblastsBasophilicproerythroblastsPolychromaticerythroblastsOrthochromaticnormoblastsMyeloblastsNeutrophilicpromyelocytesNeutrophilicmyelocytesNeutrophilicmetamyelocytesBand...
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Atlas of Clinical Hematology - part 3 pps
... Marrow IVFig. 35 a –d. Effect of hematopoieticgrowth factorsa Peripheral blood smear after the ad-ministration of G-CSFb Left shift following G-CSFc Maximum leukocyte alkaline phos-phatase ... following G-CSFd Bone marrow following chemotherapyand the administration of G-CSF de-monstrates promyelocytes and myelo-cytes, some showing mild atypia1 13 5 · Bone Marrow IVFig. 36 a –h. ... addi-tion, Heinz body formation is characteristic of a number of enzymopenic HA, and methemoglo-bin is increased in toxic HA.Several groups of hemolytic anemias are re-cognized on the basis of...
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Atlas of Clinical Hematology - part 4 potx
... andpaucity of fat cellsd Histologic section during chronicphase of CML. Naphthol AS-D chloro-acetate esterase reaction shows an ex-treme increase in neutrophilic granulo-cytopoiesis 148 Chapter ... BCR-ABL translocation, the demonstra-tion of low or even negative leukocyte alkalinephosphatase (LAP, see p. 13) w as of key impor-tance. Today the diagnosis is established by detec-tion of ... the cytogenetic detec-tion of the (9;22) translocation or the moleculargenetic detection of the BCR-ABL translocation.The absence of these changes preclude s a diagno-sis of CML (CGL).Almost...
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Atlas of Clinical Hematology - part 5 pdf
... ne-cessary for an accurate classification189 5 · Bone Marrow IVFig. 66 i Partial karyotype of a dicentricchromosome 5; 17 that leads to asimultaneous loss of the long arm of chromosome 5 ... the short arm of chromosome 17. The loss of the shortarm of chromosome 17 (17p-) causesthe loss of an allele of p53, which islocalized there in the band 17p13.Structural cha nges of 17p are ... irondeposition in the cytoplasm of a plasmacell in RARS. Nucleus is at lower right.c–f The 5q-minus syndromec Two megakaryocytes with round nu-clei and different stages of cytoplasmicmaturity –...
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Atlas of Clinical Hematology - part 6 doc
... cyto-plasm and, above the nucleus, a signal(blue-green dot at lower right) from thenormal chromosome 16 and a two -part signal (two groups of small dots at left)from the inverted chromosome 16. ... Variants of the M4 sub-typea Severely dysplastic erythropoiesis. Atcenter are three erythroblasts, two of which are very immature forms. The cy-toplasm of the upper erythroblast is va-cuolatedb ... theerythroblasts of the same patient. Thisfinding should not be mistaken for T-cellALLf PAS reaction in the bone marrow of thesame patientg Extremely immature form of M6a.Some of the cells...
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Atlas of Clinical Hematology - part 7 ppt
... subtype of ALL violates the dogma that cytoplas-mic granules are suggestive of AML.This case is a B-lineage type of ALL withcytoplasmic granules that are peroxi-dase-negative and behave as lyso-somesa ... isolated Lan-ghans giant cells Fig. 138 a, b), and toxoplasmosis(epitheloid-cell lymphadenitis of Piringer-Ku-chinka, see Fig. 136 a – d).In the examination of cervical lymph node as-pirates, ... classification of acuteleukemias. European Group for theImmunological Characterization of Leukemias (EGIL). Leukemia. 9: 178 3– 178 6]. These types of leukemia mostcommonly occur in early T-ALL withmyeloid...
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Atlas of Clinical Hematology - part 8 potx
... koăn-nen B- und T-Zell-Lymphome unterschieden wer-den. B-Zell-Lymphome sind in Europa und Ame-rika haăufiger, waăhrend die T-Zell-Lymphome inAsien uăberwiegen. Unter Einbeziehung von mor-phologischen ... Immunphaănotyp oder gene-tisch-molekulargenetisches Profil nicht moăglich.B-, T- und NK-Zell-Neoplasien werden weiterin Vorstufen-(Precursor) und reife (mature) Er-krankungen unterteilt. ... Befunden, Im-munphaănotyp, Zytogenetik und Molekulargene-tik sowie klinischen Daten hat die WHO-Arbeits-gruppe nach den Prinzipien der Revised Euro-pean-American Classification of Lymphoid Neo-plasms...
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Atlas of Clinical Hematology - part 10 pptx
... is sig-nificant lymphadenitis with clinical manifesta-tions such as headache, low-grade fever, and sin-gle or multiple lymph node enlargement, particu-larly in the neck. Involvement of the ... the rest of the patient’s life, sig-nifying that the infection has entered its latentstage. Recently, the so-called test of avidityproved to be of help in calculating the duration of infection ... Important Causative Organisms of Tropical Diseases VIIFig. 195 a – d. Filariasisa Microfilaria of Mansonella perstans, seealso Fig. 196b High-power view of a Loa loa micro-filaria with granules...
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ABC OF CLINICAL GENETICS - PART 5 pdf
... 10 .5 Frequency of cystic fibrosis mutations screenedin the North-West of EnglandMutation Frequency (%)G85E 0.3R117H 0.7621 ϩ 1G→T 1.01078delT 0.1⌬I507 0 .5 ⌬F508 88.0171 7-1 G→T 0.3G542X ... haematuriaRenal failureacg-10 11/20/01 7:27 PM Page 53 ABC of Clinical Genetics 52 Hypertrophic cardiomyopathy (HOCM) has an incidence of about 1 in 1000. Presentation is with hypertrophy of the leftand/or ... Pedigree demonstrating X linked recessive inheritance of Haemophilia Aacg-10 11/20/01 7:27 PM Page 52 ABC of Clinical Genetics 54 Box 10.9 Examples of autosomal dominant eyedisordersã Late onset...
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